Search results for "cytokine production"

showing 4 items of 4 documents

T cells involved in psoriasis vulgaris belong to the Th1 subset

1994

Although the pathogenesis of psoriasis vulgaris is still unknown, several characteristics point to an immunologically mediated process. Epidermal psoriatic lesions are characterized by a hyperproliferation of keratinocytes and an infiltration of T lymphocytes and granulocytes. Because the former may be mediated in part by lymphokines secreted by T cells, we have focused our interest on the in vivo and in vitro cytokine secretion patterns of T lymphocytes from psoriatic lesions. In five patients T lymphocytes were obtained from epidermal specimens. The cells were propagated with lectin and irradiated feeder cells and subsequently cloned by limiting dilution. The resulting T-cell clones were …

AdultMaleBiopsyCD8 AntigensT-Lymphocytesmedicine.medical_treatmentMolecular Sequence DataDermatologyBiologyPolymerase Chain ReactionBiochemistryInterferon-gammaT-Lymphocyte SubsetsPsoriasisSynovial FluidmedicineHumansPsoriasisRNA MessengerMolecular BiologyCells CulturedAgedSkinAged 80 and overBase SequenceTumor Necrosis Factor-alphaArthritis PsoriaticLymphokineInterleukinT-Lymphocytes Helper-InducerCell BiologyT lymphocyteMiddle Agedmedicine.diseaseInterleukin-10PhenotypeCytokineepidermal T cellsCD4 AntigensImmunologyCytokinesInterleukin-2cytokine productionFemaleCytokine secretionTumor necrosis factor alphaInterleukin-4CD8
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The NFκB-inducing kinase is essential for the developmental programming of skin-resident and IL-17-producing γδ T cells

2015

γδ T cells contribute to first line immune defense, particularly through their ability for rapid production of proinflammatory cytokines. The cytokine profile of γδ T cells is hard-wired already during thymic development. Yet, the molecular pathways underlying this phenomenon are incompletely understood. Here we show that signaling via the NFκB-inducing kinase (NIK) is essential for the formation of a fully functional γδ T cell compartment. In the absence of NIK, development of Vγ5+ dendritic epidermal T cells (DETCs) was halted in the embryonic thymus, and impaired NIK function caused a selective loss of IL-17 expression by γδ T cells. Using a novel conditional mutant of NIK, we could show…

MouseT-Lymphocytes10263 Institute of Experimental ImmunologyInterleukin 210302 clinical medicineT-Lymphocyte Subsets2400 General Immunology and MicrobiologyCytotoxic T cellIL-2 receptorBiology (General)0303 health sciencesGeneral NeuroscienceZAP70Interleukin-17QR2800 General NeuroscienceCell DifferentiationReceptors Antigen T-Cell gamma-deltaGeneral MedicineNatural killer T cell3. Good healthCell biologymedicine.anatomical_structureMedicineSignal TransductionResearch ArticleQH301-705.5T cellScienceImmunology610 Medicine & healthProtein Serine-Threonine KinasesBiologyγδ T cellsGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences1300 General Biochemistry Genetics and Molecular BiologymedicineAnimalsAntigen-presenting cell030304 developmental biologyGeneral Immunology and MicrobiologyNIKT cell developmentT cell cytokine productionthymic stromaMice Inbred C57BLDevelopmental Biology and Stem CellsImmunology570 Life sciences; biology030215 immunologyeLife
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Inflammatory Response to a High Radiation Dose in Breast Cancer

2014

Background: Radiation therapy (RT) is an essential treatment modality used for breast cancer (BC) care. Radiations can stimulate the immune system via the early activation of cytokine cascades, which can greatly affect cellular radio-sensitivity. Our aim is to analyze inflammatory response induced by high ionizing radiation (IR) doses, generated by intraoperative radiotherapy (IORT) treatment, to identify several potential targets that may influence cell radio-response. Methods: MCF10, MCF7 and MDA-MB231 BC cell lines have been exposed to high IR (23 Gray and 9 Gray), delivered by IORT treatments. Conditioned media (CM) were assayed at the time points: 0’, 30’, 1, 3, 6, 24, 48, and 72 hours…

Settore MED/05 - Patologia ClinicaBreast cancer IORT cytokine production
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Adrb3 adrenergic receptor is a key regulator of human myometrial apoptosis and inflammation during chorioamnionitis1

2008

The pathophysiology underlying preterm labor triggered by inflammatory conditions such as chorioamnionitis remains largely unclear. It has already been suggested that beta-3 adrenergic (ADRB3) agonists might be of interest in the pharmacological management of preterm labor. Although there is evidence implicating ADRB receptors in the control of inflammation, there are minimal data relating specifically to ADRB3. To explore the cellular consequences of chorioamnionitis and detect apoptosis, we first performed immunostaining and Western blot experiments on human myometrial samples obtained from women with confirmed chorioamnionitis. We then developed an in vitro model of chorioamnionitis by i…

preterm labormedicine.medical_specialtymedicine.medical_treatmentunited-statesbeta-adrenoceptorsStimulationInflammationin-vitroChorioamnionitisProinflammatory cytokineimmunology03 medical and health sciences0302 clinical medicineInternal medicinemedicineInterleukin 8Interleukin 6030304 developmental biology0303 health sciencesbiologycell apoptosislipopolysaccharideapoptosisbacterial infectionCell BiologyGeneral Medicinemedicine.diseasegene-expressioncytokines3. Good healthEndocrinologyCytokineReproductive Medicineinflammation030220 oncology & carcinogenesisbiology.proteincytokine productionbeta(3)-adrenoceptorTumor necrosis factor alphapregnancymedicine.symptomdeliverytumor-necrosis-factorterm
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